Aggravation of Acute Kidney Injury by Mpges-2 Down Regulation Is Associated with Autophagy Inhibition and Enhanced Apoptosis 继续阅读Aggravation of Acute Kidney Injury by Mpges-2 Down Regulation Is Associated with Autophagy Inhibition and Enhanced Apoptosis
Mir-1244 Sensitizes the Resistance of Non-Small Cell Lung Cancer A549 Cell to Cisplatin 继续阅读Mir-1244 Sensitizes the Resistance of Non-Small Cell Lung Cancer A549 Cell to Cisplatin
Resveratrol Ameliorates Oxidative Stress and Inhibits Aquaporin 4 Expression Following Rat Cerebral Ischemia-Reperfusion Injury 继续阅读Resveratrol Ameliorates Oxidative Stress and Inhibits Aquaporin 4 Expression Following Rat Cerebral Ischemia-Reperfusion Injury
Melatonin Induces Cell Apoptosis in Ags Cells through the Activation of Jnk and P38 Mapk and the Suppression of Nuclear Factor-Kappa B: A Novel Therapeutic Implication for Gastric Cancer 继续阅读Melatonin Induces Cell Apoptosis in Ags Cells through the Activation of Jnk and P38 Mapk and the Suppression of Nuclear Factor-Kappa B: A Novel Therapeutic Implication for Gastric Cancer
The Protective Role of Isorhamnetin on Human Brain Microvascular Endothelial Cells from Cytotoxicity Induced by Methylglyoxal and Oxygen-Glucose Deprivation 继续阅读The Protective Role of Isorhamnetin on Human Brain Microvascular Endothelial Cells from Cytotoxicity Induced by Methylglyoxal and Oxygen-Glucose Deprivation
Dendritic Cell-Mediated Delivery of Doxorubicin-Polyglycerol-Nanodiamond Composites Elicits Enhanced Anti-Cancer Immune Response in Glioblastoma 继续阅读Dendritic Cell-Mediated Delivery of Doxorubicin-Polyglycerol-Nanodiamond Composites Elicits Enhanced Anti-Cancer Immune Response in Glioblastoma
Mir-9-5p Promotes Msc Migration by Activating Β-Catenin Signaling Pathway 继续阅读Mir-9-5p Promotes Msc Migration by Activating Β-Catenin Signaling Pathway
Gli-1/Pi3k/Akt/Nf-Kb Pathway Mediates Resistance to Radiation and Is a Target for Reversion of Responses in Refractory Acute Myeloid Leukemia Cells 继续阅读Gli-1/Pi3k/Akt/Nf-Kb Pathway Mediates Resistance to Radiation and Is a Target for Reversion of Responses in Refractory Acute Myeloid Leukemia Cells
